Author(s): Sami Benli, Habip Sarı and Deniz Tikici

Abstract: Background: Acute pancreatitis develops as a result of the premature activation of pancreatic enzymes and manifests as abdominal pain and elevated pancreatic enzymes. The causes of acute pancreatitis are most frequently gallstones and alcohol, but another cause can be hypertriglyceridemia. Hypertriglyceridemia is responsible for approximately 1–4% of acute pancreatitis cases; serum triglyceride levels over 1000 mg/dL may trigger an attack of acute pancreatitis. Acute pancreatitis may develop when triglyceride levels reach 500–1000 mg/dL. Case presentation: Colonic ileus was detected as a result of advanced radiological examinations in a 61-year-old male patient who presented to the emergency department with complaints of abdominal pain, abdominal swelling, gas, stool, and colonic tumor was considered as a preliminary diagnosis. There are familial episodes of pancreatitis due to type 4 hyperlipidemia in the patient's past medical history. As a result of expansion, it was observed that inflammation due to pacreatic necrosis constricted the transverse distal of the colon. Subtotal colectomy, end ileostomy and pancretatic debridement were performed on the patient. The patient was discharged on the 8th postoperative day with approximately 40 cc pancreatic fistula. Conclusion: AP is a life-threatening inflammatory disorder of the pancreas which can be reversed. Although HTGP is the third most common form of pancreatitis after alcohol and gall stones, overall this disorder is quite rare. This results in the available information being sparse and the limited number of patients in the studies not allowing for generalization. Due to the absence of specific treatment guidelines for HTG AP, the HTGP patients are treated similarly to AP patients of other etiologies.

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